Final answer:
Viruses that utilize the lytic life cycle for replication usually do not induce tumors because they lyse the host cells rather than integrate their DNA into the host genome. Tumor formation is more associated with the lysogenic cycle, where viral DNA can potentially disrupt genes controlling cell division.
Step-by-step explanation:
During the lytic cycle, viruses replicate inside the host cell and then cause the cell to burst (lyse), releasing new virus particles. This lytic process does not involve the integration of viral DNA into the host's genome in a way that could cause uncontrolled cell division, which is characteristic of tumor formation. In contrast, during the lysogenic cycle, viral DNA is incorporated into the host cell's genome, existing in a dormant state known as a prophage. This can potentially lead to tumor formation if the viral DNA integrates near a gene that controls cell growth or division and disrupts its normal function. Therefore, viruses that utilize the lytic cycle for replication would typically not be involved in tumor induction, whereas viruses that establish a lysogenic cycle may have the potential to induce tumors if conditions lead to the activation of oncogenes or inactivation of tumor suppressor genes.