Final answer:
Satiety cues at the end of a meal include increased blood glucose levels, passage of food through the gastrointestinal tract, and the release of leptin from fat cells, all sending signals to the brain to induce a feeling of fullness.
Step-by-step explanation:
Satiety cues at the end of feeding include physiological responses that signal fullness and satisfaction, causing eating behavior to stop. As blood glucose levels rise, the pancreas and liver send signals to shut off hunger. The gastrointestinal tract provides satiety signals as food passes through it. Additionally, fat cells release the hormone leptin, which is crucial for signaling satiety. The hypothalamus region of the brain plays a central role in controlling the signals of hunger and satiety, responding more effectively to foods rich in fatty acids than those rich only in carbohydrates.
This regulation is sometimes referred to as a positive feedback loop, where the direction of the stimulus related to hunger changes in response to a signal indicating fullness. Consequently, the body's ability to regulate food intake is both complex and multifaceted, involving a variety of biological signals and feedback mechanisms.